全部 标题 作者
关键词 摘要

OALib Journal期刊
ISSN: 2333-9721
费用:99美元

查看量下载量

相关文章

更多...

Preoperative Screening and Case Cancellation in Cocaine-Abusing Veterans Scheduled for Elective Surgery

DOI: 10.1155/2013/149892

Full-Text   Cite this paper   Add to My Lib

Abstract:

Background. Perioperative management of cocaine-abusing patients scheduled for elective surgery varies widely based on individual anecdotes and personal experience. Methods. Chiefs of the anesthesia departments in the Veterans Affairs (VA) health system were surveyed to estimate how often they encounter surgical patients with cocaine use. Respondents were asked about their screening criteria, timing of screening, action resulting from positive screening, and if they have a formal policy for management of these patients. Interest in the development of VA guidelines for the perioperative management of patients with a history of cocaine use was also queried. Results. 172 VA anesthesia departments’ chiefs were surveyed. Response rate was 62%. Over half of the facilities see cocaine-abusing patients at least once a week (52%). Two thirds of respondents canceled or delayed patients with a positive screen regardless of clinical symptoms. Only eleven facilities (10.6%) have a formal policy. The majority of facilities (80%) thought that having formal guidelines for perioperative management of cocaine-abusing patients would be helpful to some extent. Results. 172 VA anesthesia departments’ chiefs were surveyed. Response rate was 62%. Over half of the facilities see cocaine-abusing patients at least once a week (52%). Two thirds of respondents canceled or delayed patients with a positive screen regardless of clinical symptoms. Only eleven facilities (10.6%) have a formal policy. The majority of facilities (80%) thought that having formal guidelines for perioperative management of cocaine-abusing patients would be helpful to some extent. Conclusions. There is a general consensus that formal guidelines would be helpful. Further studies are needed to help formulate evidence-based guidelines for managing patients screening positive for cocaine prior to elective surgery. 1. Introduction The National Survey on Drug Use and Health (NSDUH) estimates that 5 million Americans are regular users of cocaine, with 6000 new users daily, and more than 30 million have tried cocaine at least once [1]. Combined data from 2004 to 2006 indicate that an annual average of??7.1% of veterans met criteria for a past year substance abuse disorder [1, 2]. More specifically, 14.1% of respondents who served in the military have a history of cocaine use [1]. Cocaine produces prolonged adrenergic stimulation by blocking the presynaptic uptake of sympathomimetic neurotransmitters, including norepinephrine, serotonin, and dopamine [3]. There are two chemical forms of cocaine: the water-soluble

References

[1]  National Survey on Drug Use and Health (NSDUH), 2012, http://www.samhsa.gov/data/NSDUH.aspx.
[2]  National Survey on Drug Use & Health, http://oas.samhsa.gov/Nhsda/2k3tabs/Sect1peTabs1to66.htm#tab1. 21a.
[3]  N. M. Elkassabany, Evidence Based Practice of Anesthesiology, Elsevier, Philadelphia, Pa, USA, 2nd edition, 2010.
[4]  P. Jatlow, “Cocaine: analysis, pharmacokinetics, and metabolic disposition,” Yale Journal of Biology and Medicine, vol. 61, no. 2, pp. 105–113, 1988.
[5]  R. T. Jones, “Pharmacokinetics of cocaine: considerations when assessing cocaine use by urinalysis,” NIDA Research Monograph, vol. 175, pp. 221–234, 1997.
[6]  A. L. Misra, P. K. Nayak, R. Bloch, and S. J. Mule, “Estimation and disposition of [3H]benzoylecgonine and pharmacological activity of some cocaine metabolites,” Journal of Pharmacy and Pharmacology, vol. 27, no. 10, pp. 784–786, 1975.
[7]  R. D. Weiss and F. H. Gawin, “Protracted elimination of cocaine metabolites in long-term, high-dose cocaine abusers,” American Journal of Medicine, vol. 85, no. 6, pp. 879–880, 1988.
[8]  S. Bhargava and R. R. Arora, “Cocaine and cardiovascular complications,” American Journal of Therapeutics, vol. 18, no. 4, pp. e95–e100, 2011.
[9]  J. E. Heffner, R. A. Harley, and S. I. Schabel, “Pulmonary reactions from illicit substance abuse,” Clinics in Chest Medicine, vol. 11, no. 1, pp. 151–162, 1990.
[10]  W. Lalouschek, P. Schnider, S. Aull et al., “Cocaine abuse—with special reference to cerebrovascular complications,” Wiener Klinische Wochenschrift, vol. 107, no. 17, pp. 516–521, 1995.
[11]  S. R. Levine, J. C. M. Brust, N. Futrell et al., “A comparative study of the cerebrovascular complications of cocaine: alkaloidal versus hydrochloride—a review,” Neurology, vol. 41, no. 8, pp. 1173–1177, 1991.
[12]  R. A. Lange, J. E. Cigarroa, and L. D. Hillis, “Theodore E. Woodward award: cardiovascular complications of cocaine abuse,” Transactions of the American Clinical and Climatological Association, vol. 115, pp. 99–114, 2004.
[13]  R. A. Lange and L. D. Hillis, “Cardiovascular complications of cocaine use,” The New England Journal of Medicine, vol. 345, no. 5, pp. 351–358, 2001.
[14]  J. M. Forrester, A. W. Steele, J. A. Waldron, and P. E. Parsons, “Crack lung: an acute pulmonary syndrome with a spectrum of clinical and histopathologic findings,” American Review of Respiratory Disease, vol. 142, no. 2, pp. 462–467, 1990.
[15]  R. P. Shannon, P. Lozano, Q. Cai, W. T. Manders, and Y.-T. Shen, “Mechanism of the systemic, left ventricular, and coronary vascular tolerance to a binge of cocaine in conscious dogs,” Circulation, vol. 94, no. 3, pp. 534–541, 1996.
[16]  G. C. Baldwin, R. Choi, M. D. Roth et al., “Evidence of chronic damage to the pulmonary microcirculation in habitual users of alkaloidal (“Crack”) cocaine,” Chest, vol. 121, no. 4, pp. 1231–1238, 2002.
[17]  M. Daras, A. J. Tuchman, and S. Marks, “Central nervous system infarction related to cocaine abuse,” Stroke, vol. 22, no. 10, pp. 1320–1325, 1991.
[18]  J. H. Jones and W. B. Weir, “Cocaine-induced chest pain,” Clinics in Laboratory Medicine, vol. 26, no. 1, pp. 127–146, 2006.
[19]  S. Chakko and R. J. Myerburg, “Cardiac complications of cocaine abuse,” Clinical Cardiology, vol. 18, no. 2, pp. 67–72, 1995.
[20]  J. L. Argo, C. C. Vick, L. A. Graham, K. M. F. Itani, M. J. Bishop, and M. T. Hawn, “Elective surgical case cancellation in the Veterans Health Administration system: identifying areas for improvement,” American Journal of Surgery, vol. 198, no. 5, pp. 600–606, 2009.
[21]  E. L. Granite, N. J. Farber, and P. Adler, “Parameters for treatment of cocaine-positive patients,” Journal of Oral and Maxillofacial Surgery, vol. 65, no. 10, pp. 1984–1989, 2007.
[22]  A. K. Jha, C. M. Desroches, E. G. Campbell et al., “Use of electronic health records in U.S. Hospitals,” The New England Journal of Medicine, vol. 360, no. 16, pp. 1628–1638, 2009.
[23]  F. Garcia-Bournissen, M. Moller, M. Nesterenko, T. Karaskov, and G. Koren, “Pharmacokinetics of disappearance of cocaine from hair after discontinuation of drug use,” Forensic Science International, vol. 189, no. 1–3, pp. 24–27, 2009.
[24]  R. L. DuPont and W. A. Baumgartner, “Drug testing by urine and hair analysis: complementary features and scientific issues,” Forensic Science International, vol. 70, no. 1–3, pp. 63–76, 1995.
[25]  W. C. Brogan III, R. A. Lange, D. B. Glamann, and L. D. Hillis, “Recurrent coronary vasoconstriction caused by intranasal cocaine: possible role for metabolites,” Annals of Internal Medicine, vol. 116, no. 7, pp. 556–561, 1992.
[26]  R. L. Hawks, I. J. Kopin, R. W. Colburn, and N. B. Thoa, “Norcocaine: a pharmacologically active metabolite of cocaine found in brain,” Life Sciences, vol. 15, no. 12, pp. 2189–2195, 1974.
[27]  W. J. Crumb Jr. and C. W. Clarkson, “Characterization of the sodium channel blocking properties of the major metabolites of cocaine in single cardiac myocytes,” Journal of Pharmacology and Experimental Therapeutics, vol. 261, no. 3, pp. 910–917, 1992.
[28]  C. W. Schindler, J.-W. Zheng, and S. R. Goldberg, “Effects of cocaine and cocaine metabolites on cardiovascular function in squirrel monkeys,” European Journal of Pharmacology, vol. 431, no. 1, pp. 53–59, 2001.
[29]  S. S. Liu, R. M. Forrester, G. S. Murphy, K. Chen, and R. Glassenberg, “Anaesthetic management of a parturient with myocardial infarction related to cocaine use,” Canadian Journal of Anaesthesia, vol. 39, no. 8, pp. 858–861, 1992.
[30]  J. C. Livingston, B. C. Mabie, and J. Ramanathan, “Crack cocaine, myocardial infarction, and troponin I levels at the time of cesarean delivery,” Anesthesia and Analgesia, vol. 91, no. 4, pp. 913–915, 2000.
[31]  K. M. Kuczkowski, “Crack cocaine-induced long QT interval syndrome in a parturient with recreational cocaine use,” Annales Francaises d'Anesthesie et de Reanimation, vol. 24, no. 6, pp. 697–698, 2005.
[32]  K. M. Kuczkowski, “Crack cocaine as a cause of acute postoperative pulmonary edema in a pregnant drug addict,” Annales Francaises d'Anesthesie et de Reanimation, vol. 24, no. 4, pp. 437–438, 2005.
[33]  D. A. Vagts, C. Boklage, and C. Galli, “Intraoperative ventricular fibrillation in a patient with chronic cocaine abuse—a case report,” Anaesthesiologie und Reanimation, vol. 29, no. 1, pp. 19–24, 2004.
[34]  C. M. Bernards and A. Teijeiro, “Illicit cocaine ingestion during anesthesia,” Anesthesiology, vol. 84, no. 1, pp. 218–220, 1996.
[35]  M. D. Basson and T. Butler, “Evaluation of operating room suite efficiency in the Veterans Health Administration system by using data-envelopment analysis,” American Journal of Surgery, vol. 192, no. 5, pp. 649–656, 2006.
[36]  M. D. Basson, T. W. Butler, and H. Verma, “Predicting patient nonappearance for surgery as a scheduling strategy to optimize operating room utilization in a Veterans' Administration Hospital,” Anesthesiology, vol. 104, no. 4, pp. 826–834, 2006.
[37]  “Bureau of labor statistics, US Department of labor,” 2012, http://www.bls.gov/data/inflation_calculator.htm.
[38]  F. Dexter, J. T. Blake, D. H. Penning, and D. A. Lubarsky, “Calculating a potential increase in hospital margin for elective surgery by changing operating room time allocations or increasing nursing staffing to permit completion of more cases: a case study,” Anesthesia and Analgesia, vol. 94, no. 1, pp. 138–142, 2002.
[39]  F. Dexter, E. Marcon, R. H. Epstein, and J. Ledolter, “Validation of statistical methods to compare cancellation rates on the day of surgery,” Anesthesia and Analgesia, vol. 101, no. 2, pp. 465–473, 2005.
[40]  G. E. Hill, B. O. Ogunnaike, and E. R. Johnson, “General anaesthesia for the cocaine abusing patient. Is it safe?” British Journal of Anaesthesia, vol. 97, no. 5, pp. 654–657, 2006.
[41]  G. E. Ryb and C. Cooper, “Outcomes of cocaine-positive trauma patients undergoing surgery on the first day after admission,” The Journal of Trauma, vol. 65, no. 4, pp. 809–812, 2008.
[42]  P. Hadjizacharia, D. J. Green, D. Plurad et al., “Cocaine use in trauma: effect on injuries and outcomes,” The Journal of Trauma, vol. 66, no. 2, pp. 491–494, 2009.
[43]  P. G. Barash, C. J. Kopriva, and R. Langou, “Is cocaine a sympathetic stimulant during general anesthesia?” Journal of the American Medical Association, vol. 243, no. 14, pp. 1437–1439, 1980.

Full-Text

comments powered by Disqus

Contact Us

service@oalib.com

QQ:3279437679

WhatsApp +8615387084133

WeChat 1538708413