Interpreting the Mechanism of APOE (p.Leu167del) Mutation in the Incidence of Familial Hypercholesterolemia; An In-silico Approach

DOI: 10.2174/1874192401711010084

Keywords: Apolipoprotein E (APOE), P. Leu167del, Familial Hypercholesterolemia (FH), Homology modeling, Structural deviation, Molecular Docking Simulation

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